I came across this story written by a friend about a national scandal that I consider raises issues of concern about the provision of housing for vulnerable groups and Labour failures. According to a report by independent peer Lord Kerslake, "The Kerslake Review", Labour controlled Lambeth council has wasted £25 million funding a failed private housing company, Homes for Lambeth.
Worse, it appears at least one senior executive on a choice salary, Paul Simpson, was a former Labour Party staffer who made a career jump from one-man-band communications consultant to Head of Communications and Operations. Simpson's extensive LinkedIn profile takes credit for a number of areas which Kerslake said were failing.
The author, Sam Smith of Matthew Hopkins News has written a lengthy and careful in-depth article. As well as raising the Kerslake Review, he raised historic issues since he worked with Simpson many years ago, which of course only he knows about, but which are consistent at least with the more recent concerns. You can find the Kerslake Review itself here
It does raise ongoing concerns about the appointment of not-optimally-qualified former Labour Party staff in Lambeth and other London Boroughs.
People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.
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