I made the mistake recently of allowing Windows to update the operating system. Immediately after updating the operating system Chrome stopped logging in automatically (the bit on the top right of Chrome said "paused").
This has happened to quite a few people so I looked up to find out solutions one place that has solutions is https://9to5google.com/2020/06/15/google-chrome-windows-10-may-update-issues/.
On that I did try the Windows-L to lock and unlock, but that appeared to help sometimes, but was not that helpful.
However, I do now I think have a solution. It appears that after you login to the windows operating system it does some security processes. If you wait long enough by making a cup of tea before logging into chrome then chrome logs in OK. However, if you go immediately into chrome it fails to log in.
This is obviously some form of bug in Windows, but I am pleased that I have a solution that so far seems to work. It is also a solution that involves making a cup of tea. Hence it is a relatively efficient solution.
I have found sometimes that Chrome still loses its security access, but for now if you keep Chrome active it seems to stay connected. This is, of course, really important as if the Chrome account is "paused" everything else that needs security cannot log in. This is particularly a nuisance for things that also have an OTP which means getting loads of OTPs.
I hope people find this useful.
People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.
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