Skip to main content

BY ELECTION RESULTS 17TH JULY 2008

Derwentside DC, Castleside
Ind 297 (82.3 +8.3)
Con 64 (17.7 +3.7)
Lab [0.0 –12.0]
Majority 233
Turnout 27.49
Ind hold
Percentage change is from 2007

Hillingdon LBC, Townfield
Lab 1031(45.3 –12.6)
LD R Chamdal 506 (22.2 +8.7)
Con 445 (19.6 –9.0)
BNP 186 (8.2 +8.2)
National Front 74 (3.3 +3.3)
Green 33 (1.5 +1.5)
Majority 525
Turnout Not known
Lab hold
Percentage change is from 2006

Redditch BC, Batchley
Con 630 (39.00 –12.4)
Lab 539 (33.4 –4.3)
BNP 299 (18.5 +18.5)
LD R Patrick Taylor 121 (7.5 –3.4)
Ind 25 (1.5 +1.5)
Majority 91
Turnout 27.59
Con gain Lab
Percentage change is from 2008

Wealden DC, Uckfield New Town
LD C J Sweetland 311 (47.4 –16.0)
Con 289 (44.1 +7.5)
UKIP 56 (8.5 +8.5)
Majority 22
Turnout 31.77
LD hold
Percentage change is from 2007

Worcestershire CC, Arrow Valley East
Con 1437 (42.2 +9.9)
Lab 1041 (30.6 –15.8)
LD D Thomas 455 (13.4 –7.9)
BNP 367 (10.8 +10.8)
Ind 103 (3.0 +3.0)
Majority 396
Turnout 19.76
Con gain Lab
Percentage change is from 2005

Comments

Anon said…
Turnout in Hillingdon was 24.97%
according to website-

http://www.hillingdon.gov.uk/index.jsp?articleid=15210

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.