Skip to main content

Written Parliamentary Questions: 26th May 2006

Connecting for Health
Q:To ask the Secretary of State for Health what the status is of the Connecting for Health IT project.(John Hemming)

A:I refer the hon. Member to the reply given on 16 May 2006, Official Report, columns 939-40W. (Caroline Flint, Minister of State, Department of Health)

NHS Finances
Q:To ask the Secretary of State for Health what formula her Department uses to determine the payment made to each primary care trust under payment by results. (John Hemming)

A:Revenue allocations are made to primary care trusts (PCTs) on the basis of the relative needs of their populations. A weighted capitation formula, calculates target shares of available resources for each PCT based on the age distribution, additional need and unavoidable geographical variations in the cost of providing services. The 2006-07 allocations have been adjusted to reflect non-recurrently the transitional arrangements for PCTs to support the implementation of payment by results. (Andy Burnham, Minister of State, Department of Health)

NHS Hospital Trusts
Q:To ask the Secretary of State for Health pursuant to the Answer of 19 April 2006, Official Report, column 743W, on NHS Hospital Trusts, if she will set out the figures for fixed costs identifying those that arise in relation to private finance initiative projects.(John Hemming)

A:I refer the hon. Member to the reply given on 12 July 2005, Official Report, columns 988-89W. The table which has been placed in the Library gives the annual payments by each trust to it's private sector partner on private finance initiative schemes which have reached financial close. This table has now been updated to reflect the Barts and London Hospital and the Hull and East Yorkshire private finance initiative schemes reaching financial close since then.
(Andy Burnham, Minister of State, Department of Health)

Comments

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.