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Melatonin - a route towards improved cellular health (reversing cellular aging)

I have recently done another video about Melatonin.

On my blog entry about insomnia I go into some links relating to Melatonin. It is clear, however, that Melatonin is a key for reducing the decay that occurs in cells. At the moment I am trying to set up a trial into the use of melatonin for improving cellular health reversing the entropic decay that occurs over time (cellular aging). If anyone is interested in participating in such a trial they should email me. melatonin site

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Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.
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