Skip to main content

Greece and the EU

It is not that easy to predict what will happen with Greece. The arguments of the Greek government are absurd. They cannot revisit the Second World War when the country agreed a settlement many years ago. The argument about profits on Greek bonds is a nonsense given the amount that has already been written off. Now we have, "if we go it falls apart".

If they renege on their contractual commitments "if falls apart".  Which is why the other EU countries won't just let them off their treaty obligations.

My personal view is that the elected representatives for Syriza are not going to compromise enough and will push it to the limits.  That is likely to lead to Greece being in default and short of cash.  In the end this is actually enough to drive them out of the EU.  They are unlikely to agree to enforce any ECJ judgments in respect of paying debt.  Hence I think they are on the way out of the EU itself - not just the Eurozone.

The fact that they have started with arguments that hold no water at all does lead to the conclusion that Grexit is the end game.  However, nothing is certain.  The difficulty with Grexit is that no-one would be enthusiastic about "new drachmas" with Syriza in charge.

Not a good situation.





Comments

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.